As regards the special _treatment_ of these conditions, the patient must, in the first case, be warned against straining of all sorts. Pneumatoceles require no other treatment. The condition soon disappears if the patient recovers. In widespread surgical emphysema, an incision should be made over the injured sinus, thus allowing of the direct escape of the air expelled from that sinus.
=The involvement of nerves.= The following nerves may be involved in anterior fossa fractures:--
(_a_) _The olfactory nerve._ The great majority of anterior fossa fractures traverse the cribriform plate, necessarily injuring the fine branches of the olfactory bulb. The bulb itself may be lacerated, with or without injury to the under surface of the frontal lobes. Sir Prescott Hewitt considered that anosmia, or loss of smell, resulted most frequently from blows applied to the back of the head, the frontal region being injured by contre-coup. From my own experience it would appear, however, that anosmia, whether uni- or bilateral, whether transient or permanent, generally results from direct injuries of the cribriform plate with associated lacerations of the olfactory nerves. It is difficult to estimate the presence or degree of immediate loss of smell on account of the general condition of the patient and because the nostrils are usually more or less filled with blood coagulum. Experience shows, however, that early loss of smell is the rule and total and permanent anosmia the exception. Anosmia is usually associated with some degree of loss of taste.
(_b_) _The optic nerve._ Many cases have been recorded in which visual defects resulted from blows applied to the head. The blindness may be partial or complete, immediate in onset or developing at some future date. In the latter case, the loss of vision is due to retinal changes or results from post-neuritic atrophy.
The occurrence of complete or partial blindness as the immediate result of the injury is, at first sight, difficult to explain, for the vast majority of anterior fossa fractures avoid the immediate vicinity of the optic foramina, passing by preference between the two foramina or diverging towards the sphenoidal fissures. Small fissured fractures not infrequently radiate through the optic foramina, usually, however, of so slight a nature as to be incapable of leading to any gross lesion of the optic nerves. Hæmorrhage into the sheath of the nerve is probably responsible for a certain proportion of cases, more especially those in which there is a peripheral concentric loss of vision, the more central fibres escaping. It is possible, also, that cases evidencing temporal or nasal blindness may be due, as J. J. Evans[17] thinks, to a contre-coup contusion of the nerve through it being forcibly driven against the bony boundaries of the foramen. Taking into consideration, however, the very frequent presence of a fracture through the anterior clinoid process (see p. 82), and the usual displacement of that process, it would appear probable that immediate and more or less complete loss of vision results from the compression and crushing of the optic nerve by reason of the pressure exercised by a displaced clinoid process.