During the course of routine examinations several transient glycosurias were seen. Their transient character was the outstanding feature. The quantity of sugar was very moderate—our figures were never above 1 per cent.—and the daily amount of urine was always within normal limits. Acetone and diacetic acid were absent. A few observations on the blood sugar showed a rise (.2 to .25), which readily came to normal with treatment. Clinically these cases were not classed as diabetes mellitus, but rather as a nervous complication of influenza, involving in some way the carbohydrate metabolism, probably through the central nervous system. One case of special interest, which is mentioned elsewhere, was the association of glycosuria with almost total blindness from a very intense optic œdema. Sugar (1 per cent.) was present on the day of admission, while only a trace was noted on the two following days, and from then on the urine was free from sugar. How many days the sugar had been present before admission to the hospital we cannot say, but we could trace the failure of vision back to almost the day of its onset, which was three weeks previous to our first examination. The eye symptoms were the only complaints. The patient had had a moderately sharp attack of influenza a little over two weeks before the first sign of failure of vision had appeared. We may add that the vision returned slowly to normal several weeks after admission. The urine and blood sugar were normal, on a general diet, over a period of one month while in the hospital. Unfortunately, we have had no further record of this patient regarding the urine, but her vision still remains normal. Cases of this type were observed in England after the 1890 epidemic, and are referred to in Allbutt’s “System of Medicine,” vol. i, on influenza. Our other glycosuria cases did not present changes in the fundus of the eye. The glycosuria and glycæmia were transient, and we feel that they do not represent diabetes mellitus. Most of the patients of this class had long since recovered from an attack of influenza, and came to the hospital usually for treatment of various nervous conditions, which at times simulated neuritis, or otherwise one saw manifestations of general nervousness, not unlike hyperthyroidism. In all probability, we were dealing with a hyperglycæmia associated with a hyperactive thyroid gland. So, after all, the glycosuria, even though rare, is not bewildering. Symptoms and signs of toxic goitre in direct relation to the epidemic we claim to have seen, and one is justified, temporarily at least, in having the thyroid gland father our transient glycosuria.